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CD36 molecule

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mammalian protein found in Homo sapiens

Risorse esterne

Ensembl protein ID
ENSP00000308165[1]
ENSP00000378268[2]
ENSP00000399421[3]
ENSP00000401863[4]
ENSP00000411411[5]
ENSP00000415743[6]
ENSP00000439543[7]
ENSP00000441956[8]
ENSP00000392298[9]
ENSP00000396258[10]
ENSP00000398760[11]
ENSP00000407690[12]
ENSP00000409762[13]
ENSP00000410371[14]
ENSP00000416388[15]
ENSP00000431296[16]
ENSP00000433659[17]
ENSP00000435698[18]
identificativo RefSeq di una proteina
NP_000063[19]
NP_001001547[19]
NP_001001548[19]
NP_001120915[19]
NP_001120916[19]
NP_001276837[19]
NP_001276838[19]
NP_001276840[19]
XP_005250772[19]
XP_024302770[19]
XP_024302771[19]
NP_001358003[19]
NP_001358004[19]
NP_001358006[19]
NP_001358007[19]
NP_001358008[19]
NP_001358009[19]
NP_001358010[19]
identificativo UniProt
P16671[20]
Transporter Classification Database ID
9.B.39.1.4[21][22]
identificativo Freebase
/m/04bkfw
identificativo OpenAlex
C2779828298[23]

no tipo 'e

proteina[20]

parte 'e

CD36[24]
Putative uncharacterized transport proteins[25]

trovato nella tassonomia de

Homo sapiens[20]

codificato da

CD36[20]

funzione molecolare

legame dei farmaci alle proteine plasmatiche[26][27][28][29]

metodo di determinazione: IPI

low-density lipoprotein particle receptor activity[30][31][32]

metodo di determinazione: TAS, IMP, IEA

transforming growth factor beta binding[33]

metodo di determinazione: ISS

high-density lipoprotein particle binding[32]

metodo di determinazione: IEA

lipoprotein particle binding[34]

metodo di determinazione: IDA

thrombospondin receptor activity[33]

metodo di determinazione: ISS

lipid binding[35]

metodo di determinazione: IDA

low-density lipoprotein particle binding[32][35]

metodo di determinazione: IDA, IEA

lipoteichoic acid immune receptor activity[32]

metodo di determinazione: IEA

amyloid-beta binding[36][37]

metodo di determinazione: IC, TAS

scavenger receptor activity[38][39][40]

metodo di determinazione: IMP, TAS

Toll-like receptor binding[41]

metodo di determinazione: IPI

oxidised low-density lipoprotein particle receptor activity[42]

metodo di determinazione: IMP

amyloid-beta binding[43][44][45]

metodo di determinazione: IC, IDA, TAS

protein-containing complex binding[46]

metodo di determinazione: IPI

componente cellulare

apparato del Golgi[47][32][48][49]

metodo di determinazione: IDA, IEA

apical plasma membrane[47]

metodo di determinazione: IEA

phagocytic vesicle[50]

metodo di determinazione: TAS

apical part of cell[32]

metodo di determinazione: IEA

brush border membrane[51][32]

metodo di determinazione: ISS, IEA

endocytic vesicle membrane[50]

metodo di determinazione: TAS

integral component of plasma membrane[52][30]

metodo di determinazione: TAS

membrana[52][47][53][32]

metodo di determinazione: TAS, IEA

external side of plasma membrane[32]

metodo di determinazione: IEA

integral component of membrane[47]

metodo di determinazione: IEA

platelet alpha granule membrane[50]

metodo di determinazione: TAS

Zattera lipidica[47][32][48]

metodo di determinazione: IDA, IEA

cell surface[54][55]

metodo di determinazione: IDA, ISS

membrana cellulare[50][47][32][56]

metodo di determinazione: IDA, TAS, IEA

spazio extracellulare[57]

metodo di determinazione: IDA

specific granule membrane[50]

metodo di determinazione: TAS

spazio extracellulare[58]

metodo di determinazione: HDA

intracellulare[59]

metodo di determinazione: IPI

cell surface[38][33][35]

metodo di determinazione: IDA, ISS, TAS

receptor complex[41]

metodo di determinazione: IPI

processo biologico

low-density lipoprotein particle mediated signaling[32]

metodo di determinazione: IEA

apoptotic cell clearance[32]

metodo di determinazione: IEA

positive regulation of MAPK cascade[32]

metodo di determinazione: IEA

response to lipid[51][32]

metodo di determinazione: ISS, IEA

nitric oxide mediated signal transduction[60]

metodo di determinazione: IDA

intestinal absorption[51][32]

metodo di determinazione: ISS, IEA

positive regulation of blood microparticle formation[32]

metodo di determinazione: IEA

positive regulation of blood coagulation[32]

metodo di determinazione: IEA

plasma lipoprotein particle clearance[61][32]

metodo di determinazione: ISS, IEA

sensory perception of taste[51][32]

metodo di determinazione: ISS, IEA

coagulazione del sangue[62]

metodo di determinazione: TAS

cellular response to hydroperoxide[32]

metodo di determinazione: IEA

positive regulation of ERK1 and ERK2 cascade[51][32]

metodo di determinazione: ISS, IEA

receptor internalization[51][32]

metodo di determinazione: ISS, IEA

cellular response to bacterial lipopeptide[32]

metodo di determinazione: IEA

amyloid fibril formation[51][32]

metodo di determinazione: ISS, IEA

cellular response to lipopolysaccharide[32]

metodo di determinazione: IEA

cell surface receptor signaling pathway[53][32]

metodo di determinazione: IEA

antigen processing and presentation of exogenous peptide antigen via MHC class I, TAP-dependent[50]

metodo di determinazione: TAS

cholesterol transport[61][32]

metodo di determinazione: ISS, IEA

platelet degranulation[50]

metodo di determinazione: TAS

cholesterol import[51][32]

metodo di determinazione: ISS, IEA

cellular response to diacyl bacterial lipopeptide[48]

metodo di determinazione: IDA

negative regulation of transcription by RNA polymerase II[32]

metodo di determinazione: IEA

trasporto[63]

metodo di determinazione: IEA

toll-like receptor TLR6:TLR2 signaling pathway[50]

metodo di determinazione: TAS

phagocytosis, recognition[32]

metodo di determinazione: IEA

cellular response to low-density lipoprotein particle stimulus[51][32]

metodo di determinazione: ISS, IEA

positive regulation of macrophage cytokine production[32]

metodo di determinazione: IEA

risposta immunitaria[53]

metodo di determinazione: IEA

response to linoleic acid[51][32]

metodo di determinazione: ISS, IEA

negative regulation of gene expression[32]

metodo di determinazione: IEA

positive regulation of reactive oxygen species metabolic process[32]

metodo di determinazione: IEA

lipoprotein transport[30][31][32]

metodo di determinazione: TAS, IMP, IEA

positive regulation of cell-matrix adhesion[60]

metodo di determinazione: IDA

positive regulation of interleukin-12 production[32]

metodo di determinazione: IEA

triglyceride transport[51][32]

metodo di determinazione: ISS, IEA

long-chain fatty acid import into cell[32][56][60]

metodo di determinazione: IDA, IEA

MyD88-dependent toll-like receptor signaling pathway[50]

metodo di determinazione: TAS

response to fatty acid[51][32]

metodo di determinazione: ISS, IEA

regulation of removal of superoxide radicals[32]

metodo di determinazione: IEA

cellular response to lipoteichoic acid[32]

metodo di determinazione: IEA

positive regulation of cholesterol storage[32]

metodo di determinazione: IEA

intestinal cholesterol absorption[51][32]

metodo di determinazione: ISS, IEA

positive regulation of tumor necrosis factor production[32]

metodo di determinazione: IEA

receptor-mediated endocytosis[64]

metodo di determinazione: TAS

lipid metabolism[65]

metodo di determinazione: NAS

cGMP-mediated signaling[60]

metodo di determinazione: IDA

positive regulation of phagocytosis, engulfment[32]

metodo di determinazione: IEA

defense response to Gram-positive bacterium[32]

metodo di determinazione: IEA

positive regulation of cytosolic calcium ion concentration[51][32]

metodo di determinazione: ISS, IEA

positive regulation of interleukin-6 production[32]

metodo di determinazione: IEA

positive regulation of peptidyl-tyrosine phosphorylation[32]

metodo di determinazione: IEA

positive regulation of I-kappaB kinase/NF-kappaB signaling[32]

metodo di determinazione: IEA

response to stilbenoid[32]

metodo di determinazione: IEA

positive regulation of NLRP3 inflammasome complex assembly[51][32]

metodo di determinazione: ISS, IEA

low-density lipoprotein particle clearance[31][32]

metodo di determinazione: IEA, IMP

lipid storage[31][32]

metodo di determinazione: IEA, IMP

positive regulation of macrophage derived foam cell differentiation[31][32]

metodo di determinazione: IEA, IMP

cell adhesion[62][47]

metodo di determinazione: IEA, TAS

toll-like receptor signaling pathway[50]

metodo di determinazione: TAS

neutrophil degranulation[50]

metodo di determinazione: TAS

regulation of lipid metabolic process[50]

metodo di determinazione: TAS

production of molecular mediator involved in inflammatory response[38]

metodo di determinazione: TAS

metabolismo dei lipidi[38]

metodo di determinazione: TAS

receptor-mediated endocytosis[50][40]

metodo di determinazione: IMP, TAS

phagocytosis, engulfment[38]

metodo di determinazione: TAS

response to bacterium[32]

metodo di determinazione: IEA

positive regulation of gene expression[66][32]

metodo di determinazione: IEA, ISS

positive regulation of cell death[42]

metodo di determinazione: IMP

cytokine-mediated signaling pathway[50]

metodo di determinazione: TAS

regulation of lipopolysaccharide-mediated signaling pathway[38]

metodo di determinazione: TAS

regulation of toll-like receptor signaling pathway[67]

metodo di determinazione: IGI

negative regulation of protein import into nucleus[32]

metodo di determinazione: IEA

regulation of protein heterodimerization activity[68]

metodo di determinazione: IMP

positive regulation of nitric oxide biosynthetic process[66][32]

metodo di determinazione: IEA, ISS

positive regulation of NF-kappaB transcription factor activity[67]

metodo di determinazione: IGI

energy homeostasis[51][32]

metodo di determinazione: IEA, ISS

regulation of action potential[66][32]

metodo di determinazione: IEA, ISS

positive regulation of cold-induced thermogenesis[69][32]

metodo di determinazione: IEA, ISS

cellular response to oxidised low-density lipoprotein particle stimulus[42]

metodo di determinazione: IMP

oxidised low-density lipoprotein particle clearance[42]

metodo di determinazione: IMP

amyloid-beta clearance by cellular catabolic process[40]

metodo di determinazione: IMP

positive regulation of reactive oxygen species biosynthetic process[66][32]

metodo di determinazione: IEA, ISS

cellular response to amyloid-beta[67]

metodo di determinazione: IGI

immagine Gene Atlas

categoria su Commons

Fatty taste receptors

Riferimento

  1. ↑ Ensembl Release 99, ENSP00000308165
  2. ↑ Ensembl Release 99, ENSP00000378268
  3. ↑ Ensembl Release 99, ENSP00000399421
  4. ↑ Ensembl Release 99, ENSP00000401863
  5. ↑ Ensembl Release 99, ENSP00000411411
  6. ↑ Ensembl Release 99, ENSP00000415743
  7. ↑ Ensembl Release 99, ENSP00000439543
  8. ↑ Ensembl Release 99, ENSP00000441956
  9. ↑ Ensembl Release 99, ENSP00000392298
  10. ↑ Ensembl Release 99, ENSP00000396258
  11. ↑ Ensembl Release 99, ENSP00000398760
  12. ↑ Ensembl Release 99, ENSP00000407690
  13. ↑ Ensembl Release 99, ENSP00000409762
  14. ↑ Ensembl Release 99, ENSP00000410371
  15. ↑ Ensembl Release 99, ENSP00000416388
  16. ↑ Ensembl Release 99, ENSP00000431296
  17. ↑ Ensembl Release 99, ENSP00000433659
  18. ↑ Ensembl Release 99, ENSP00000435698
  19. ↑ 19,00 19,01 19,02 19,03 19,04 19,05 19,06 19,07 19,08 19,09 19,10 19,11 19,12 19,13 19,14 19,15 19,16 19,17 NCBI Gene, 30 Maj 2020, 948
  20. ↑ 20,0 20,1 20,2 20,3 UniProt, 13 Nuv 2019, P16671
  21. ↑ Classificazione TC, 13 Màr 2020
  22. ↑ Classificazione TC, 29 Abb 2020
  23. ↑ OpenAlex, 26 Jen 2022, https://docs.openalex.org/download-snapshot/snapshot-data-format
  24. ↑ InterPro Release 71.0, http://www.ebi.ac.uk/interpro/protein/P16671
  25. ↑ 9.B.39.1.4, 2 ott 2019, hierarchical database ID heuristic
  26. ↑ Membrane sorting of toll-like receptor (TLR)-2/6 and TLR2/1 heterodimers at the cell surface determines heterotypic associations with CD36 and intracellular targeting, GOA, 8 Abb 2019, P16671, UniProt, http://www.ebi.ac.uk/QuickGO/annotations?geneProductId=UniProtKB:P16671, IPI
  27. ↑ IntAct protein interaction database, 8 Abb 2019, Molecular basis of antiangiogenic thrombospondin-1 type 1 repeat domain interactions with CD36, GOA, IPI, http://www.ebi.ac.uk/QuickGO/annotations?geneProductId=UniProtKB:P16671
  28. ↑ IntAct protein interaction database, 8 Abb 2019, CD36 ligands promote sterile inflammation through assembly of a Toll-like receptor 4 and 6 heterodimer, GOA, IPI, http://www.ebi.ac.uk/QuickGO/annotations?geneProductId=UniProtKB:P16671
  29. ↑ Critical Assessment of Protein Function Annotation, 8 Abb 2019, The Cysteine-Rich Interdomain Region from the Highly Variable Plasmodium falciparum Erythrocyte Membrane Protein-1 Exhibits a Conserved Structure, GOA, IPI, http://www.ebi.ac.uk/QuickGO/annotations?geneProductId=UniProtKB:P16671
  30. ↑ 30,0 30,1 30,2 British Heart Foundation, 8 Abb 2019, PPARgamma promotes monocyte/macrophage differentiation and uptake of oxidized LDL, GOA, TAS, http://www.ebi.ac.uk/QuickGO/annotations?geneProductId=UniProtKB:P16671
  31. ↑ 31,0 31,1 31,2 31,3 31,4 British Heart Foundation, 8 Abb 2019, Macrophage scavenger receptor CD36 is the major receptor for LDL modified by monocyte-generated reactive nitrogen species, GOA, IMP, http://www.ebi.ac.uk/QuickGO/annotations?geneProductId=UniProtKB:P16671
  32. ↑ 32,00 32,01 32,02 32,03 32,04 32,05 32,06 32,07 32,08 32,09 32,10 32,11 32,12 32,13 32,14 32,15 32,16 32,17 32,18 32,19 32,20 32,21 32,22 32,23 32,24 32,25 32,26 32,27 32,28 32,29 32,30 32,31 32,32 32,33 32,34 32,35 32,36 32,37 32,38 32,39 32,40 32,41 32,42 32,43 32,44 32,45 32,46 32,47 32,48 32,49 32,50 32,51 32,52 32,53 32,54 32,55 32,56 32,57 32,58 32,59 32,60 32,61 32,62 32,63 32,64 Ensembl, 8 Abb 2019, GOA, IEA, http://www.ebi.ac.uk/QuickGO/annotations?geneProductId=UniProtKB:P16671
  33. ↑ 33,0 33,1 33,2 British Heart Foundation, 8 Abb 2019, GOA, ISS, http://www.ebi.ac.uk/QuickGO/annotations?geneProductId=UniProtKB:P16671
  34. ↑ PPARgamma promotes monocyte/macrophage differentiation and uptake of oxidized LDL, GOA, 8 Abb 2019, P16671, UniProt, http://www.ebi.ac.uk/QuickGO/annotations?geneProductId=UniProtKB:P16671, IDA
  35. ↑ 35,0 35,1 35,2 British Heart Foundation, 8 Abb 2019, Macrophage scavenger receptor CD36 is the major receptor for LDL modified by monocyte-generated reactive nitrogen species, GOA, IDA, http://www.ebi.ac.uk/QuickGO/annotations?geneProductId=UniProtKB:P16671
  36. ↑ ARUK-UCL, 1 Jen 2018, CD36 ligands promote sterile inflammation through assembly of a Toll-like receptor 4 and 6 heterodimer, GOA, TAS, http://www.ebi.ac.uk/QuickGO/annotations?protein=P16671&geneProductId=UniProtKB:P16671
  37. ↑ ARUK-UCL, 1 Jen 2018, CD36 ligands promote sterile inflammation through assembly of a Toll-like receptor 4 and 6 heterodimer, GOA, IC, http://www.ebi.ac.uk/QuickGO/annotations?protein=P16671&geneProductId=UniProtKB:P16671
  38. ↑ 38,0 38,1 38,2 38,3 38,4 38,5 ARUK-UCL, 8 Abb 2019, CD36 Deficiency Suppresses Epileptic Seizures, GOA, TAS, http://www.ebi.ac.uk/QuickGO/annotations?geneProductId=UniProtKB:P16671
  39. ↑ ARUK-UCL, 8 Abb 2019, Scavenger receptor class B type I (SR-BI) mediates adhesion of neonatal murine microglia to fibrillar beta-amyloid, GOA, TAS, http://www.ebi.ac.uk/QuickGO/annotations?geneProductId=UniProtKB:P16671
  40. ↑ 40,0 40,1 40,2 ARUK-UCL, 8 Abb 2019, IL-4-induced selective clearance of oligomeric beta-amyloid peptide(1-42) by rat primary type 2 microglia., GOA, IMP, http://www.ebi.ac.uk/QuickGO/annotations?geneProductId=UniProtKB:P16671
  41. ↑ 41,0 41,1 ARUK-UCL, 8 Abb 2019, CD36 ligands promote sterile inflammation through assembly of a Toll-like receptor 4 and 6 heterodimer, GOA, IPI, http://www.ebi.ac.uk/QuickGO/annotations?geneProductId=UniProtKB:P16671
  42. ↑ 42,0 42,1 42,2 42,3 ARUK-UCL, 8 Abb 2019, Oxidized Lipoprotein Uptake Through the CD36 Receptor Activates the NLRP3 Inflammasome in Human Retinal Pigment Epithelial Cells., GOA, IMP, http://www.ebi.ac.uk/QuickGO/annotations?geneProductId=UniProtKB:P16671
  43. ↑ ARUK-UCL, 8 Abb 2019, CD36 ligands promote sterile inflammation through assembly of a Toll-like receptor 4 and 6 heterodimer, GOA, TAS, http://www.ebi.ac.uk/QuickGO/annotations?geneProductId=UniProtKB:P16671
  44. ↑ ARUK-UCL, 8 Abb 2019, TREM2 Is a Receptor for β-Amyloid that Mediates Microglial Function., GOA, IDA, http://www.ebi.ac.uk/QuickGO/annotations?geneProductId=UniProtKB:P16671
  45. ↑ ARUK-UCL, 8 Abb 2019, CD36 ligands promote sterile inflammation through assembly of a Toll-like receptor 4 and 6 heterodimer, GOA, IC, http://www.ebi.ac.uk/QuickGO/annotations?geneProductId=UniProtKB:P16671
  46. ↑ ARUK-UCL, 8 Abb 2019, TREM2 Is a Receptor for β-Amyloid that Mediates Microglial Function., GOA, IPI, http://www.ebi.ac.uk/QuickGO/annotations?geneProductId=UniProtKB:P16671
  47. ↑ 47,0 47,1 47,2 47,3 47,4 47,5 47,6 GOA, 8 Abb 2019, P16671, UniProt, http://www.ebi.ac.uk/QuickGO/annotations?geneProductId=UniProtKB:P16671, IEA
  48. ↑ 48,0 48,1 48,2 Membrane sorting of toll-like receptor (TLR)-2/6 and TLR2/1 heterodimers at the cell surface determines heterotypic associations with CD36 and intracellular targeting, GOA, 8 Abb 2019, P16671, UniProt, http://www.ebi.ac.uk/QuickGO/annotations?geneProductId=UniProtKB:P16671, IDA
  49. ↑ Human Protein Atlas, 8 Abb 2019, GOA, IDA, http://www.ebi.ac.uk/QuickGO/annotations?geneProductId=UniProtKB:P16671
  50. ↑ 50,00 50,01 50,02 50,03 50,04 50,05 50,06 50,07 50,08 50,09 50,10 50,11 50,12 50,13 Reactome, 8 Abb 2019, GOA, TAS, http://www.ebi.ac.uk/QuickGO/annotations?geneProductId=UniProtKB:P16671
  51. ↑ 51,00 51,01 51,02 51,03 51,04 51,05 51,06 51,07 51,08 51,09 51,10 51,11 51,12 51,13 51,14 51,15 GOA, 8 Abb 2019, P16671, UniProt, http://www.ebi.ac.uk/QuickGO/annotations?geneProductId=UniProtKB:P16671, ISS
  52. ↑ 52,0 52,1 Proteome Inc., 8 Abb 2019, Isolation and characterization of platelet glycoprotein IV (CD36), GOA, TAS, http://www.ebi.ac.uk/QuickGO/annotations?geneProductId=UniProtKB:P16671
  53. ↑ 53,0 53,1 53,2 InterPro, 8 Abb 2019, GOA, IEA, http://www.ebi.ac.uk/QuickGO/annotations?geneProductId=UniProtKB:P16671
  54. ↑ British Heart Foundation, 23 Maj 2017, GOA, ISS, http://www.ebi.ac.uk/QuickGO/GAnnotation?protein=P16671
  55. ↑ British Heart Foundation, 23 Maj 2017, Macrophage scavenger receptor CD36 is the major receptor for LDL modified by monocyte-generated reactive nitrogen species, GOA, IDA, http://www.ebi.ac.uk/QuickGO/GAnnotation?protein=P16671
  56. ↑ 56,0 56,1 Overexpression of CD36 and acyl-CoA synthetases FATP2, FATP4 and ACSL1 increases fatty acid uptake in human hepatoma cells, GOA, 8 Abb 2019, P16671, UniProt, http://www.ebi.ac.uk/QuickGO/annotations?geneProductId=UniProtKB:P16671, IDA
  57. ↑ Human colostrum: identification of minor proteins in the aqueous phase by proteomics, GOA, 23 Maj 2017, P16671, UniProt, http://www.ebi.ac.uk/QuickGO/GAnnotation?protein=P16671, IDA
  58. ↑ Human colostrum: identification of minor proteins in the aqueous phase by proteomics, GOA, 8 Abb 2019, P16671, UniProt, http://www.ebi.ac.uk/QuickGO/annotations?geneProductId=UniProtKB:P16671, HDA
  59. ↑ ARUK-UCL, 1 Jen 2018, CD36 ligands promote sterile inflammation through assembly of a Toll-like receptor 4 and 6 heterodimer, GOA, IPI, http://www.ebi.ac.uk/QuickGO/annotations?protein=P16671&geneProductId=UniProtKB:P16671
  60. ↑ 60,0 60,1 60,2 60,3 British Heart Foundation, 8 Abb 2019, Thrombospondin-1 inhibits nitric oxide signaling via CD36 by inhibiting myristic acid uptake, GOA, IDA, http://www.ebi.ac.uk/QuickGO/annotations?geneProductId=UniProtKB:P16671
  61. ↑ 61,0 61,1 British Heart Foundation, 8 Abb 2019, Scavenger receptors class A-I/II and CD36 are the principal receptors responsible for the uptake of modified low density lipoprotein leading to lipid loading in macrophages, GOA, ISS, http://www.ebi.ac.uk/QuickGO/annotations?geneProductId=UniProtKB:P16671
  62. ↑ 62,0 62,1 Proteome Inc., 8 Abb 2019, Identification of glycoprotein IV (CD36) as a primary receptor for platelet-collagen adhesion, GOA, TAS, http://www.ebi.ac.uk/QuickGO/annotations?geneProductId=UniProtKB:P16671
  63. ↑ GOA, 23 Maj 2017, P16671, UniProt, http://www.ebi.ac.uk/QuickGO/GAnnotation?protein=P16671, IEA
  64. ↑ Reactome, 23 Maj 2017, GOA, TAS, http://www.ebi.ac.uk/QuickGO/GAnnotation?protein=P16671
  65. ↑ Proteome Inc., 8 Abb 2019, Structural organization of the gene for human CD36 glycoprotein, GOA, NAS, http://www.ebi.ac.uk/QuickGO/annotations?geneProductId=UniProtKB:P16671
  66. ↑ 66,0 66,1 66,2 66,3 ARUK-UCL, 8 Abb 2019, GOA, ISS, http://www.ebi.ac.uk/QuickGO/annotations?geneProductId=UniProtKB:P16671
  67. ↑ 67,0 67,1 67,2 ARUK-UCL, 8 Abb 2019, CD36 ligands promote sterile inflammation through assembly of a Toll-like receptor 4 and 6 heterodimer, GOA, IGI, http://www.ebi.ac.uk/QuickGO/annotations?geneProductId=UniProtKB:P16671
  68. ↑ ARUK-UCL, 8 Abb 2019, CD36 ligands promote sterile inflammation through assembly of a Toll-like receptor 4 and 6 heterodimer, GOA, IMP, http://www.ebi.ac.uk/QuickGO/annotations?geneProductId=UniProtKB:P16671
  69. ↑ YuBioLab, 8 Abb 2019, CD36 is indispensable for thermogenesis under conditions of fasting and cold stress., GOA, ISS, http://www.ebi.ac.uk/QuickGO/annotations?geneProductId=UniProtKB:P16671
Estratto 'e "https://nap.wikipedia.org/wiki/Speciàle:AboutTopic/Q4035577"